Education and Training
- Resident, Internal Medicine, Mount Sinai School of Medicine, 2003 - 2005
- Intern, Internal Medicine, Mount Sinai School of Medicine, 2002 - 2003
- M.D., Thomas Jefferson University, 2002
Toth, K, Slosky, LM, Pack, TF, Urs, NM, Boone, P, Mao, L, Abraham, D, Caron, MG, and Barak, LS. "Ghrelin receptor antagonism of hyperlocomotion in cocaine-sensitized mice requires βarrestin-2." Synapse (New York, N.Y.) 72, no. 1 (January 2018).
Hershberger, KA, Abraham, DM, Martin, AS, Mao, L, Liu, J, Gu, H, Locasale, JW, and Hirschey, MD. "Sirtuin 5 is required for mouse survival in response to cardiac pressure overload." The Journal of biological chemistry 292, no. 48 (December 2017): 19767-19781.
Martin, AS, Abraham, DM, Hershberger, KA, Bhatt, DP, Mao, L, Cui, H, Liu, J, Liu, X, Muehlbauer, MJ, Grimsrud, PA, Locasale, JW, Payne, RM, and Hirschey, MD. "Nicotinamide mononucleotide requires SIRT3 to improve cardiac function and bioenergetics in a Friedreich's ataxia cardiomyopathy model." JCI insight 2, no. 14 (July 20, 2017).
Abraham, DM, Davis, RT, Warren, CM, Mao, L, Wolska, BM, Solaro, RJ, and Rockman, HA. "β-Arrestin mediates the Frank-Starling mechanism of cardiac contractility." Proceedings of the National Academy of Sciences of the United States of America 113, no. 50 (December 2016): 14426-14431.
Torok, RD, Abraham, DM, Gareri, C, Mao, L, and Rockman, HA. "TWIK-Related Acid-Sensitive K+ Channels Affect the Development of Aortic Root Aneurysms in Mice." November 11, 2016.
Abraham, D, and Mao, L. "Cardiac Pressure-Volume Loop Analysis Using Conductance Catheters in Mice." Journal of visualized experiments : JoVE, no. 103 (September 17, 2015).
Pironti, G, Strachan, RT, Abraham, D, Mon-Wei Yu, S, Chen, M, Chen, W, Hanada, K, Mao, L, Watson, LJ, and Rockman, HA. "Circulating Exosomes Induced by Cardiac Pressure Overload Contain Functional Angiotensin II Type 1 Receptors." Circulation 131, no. 24 (June 2015): 2120-2130.
Abraham, DM, Lee, TE, Watson, LJ, Mao, L, Chandok, G, Wolf, MJ, and Rockman, HA. "TREK-1 Modulates Fibrosis & Diastolic Dysfunction Through Activation of Stress-Activated Kinases." November 25, 2014.
Abraham, DM, and Marchuk, DA. "Inhibition of the cardiomyocyte-specific troponin I-interacting kinase limits oxidative stress, injury, and adverse remodeling due to ischemic heart disease." Circulation research 114, no. 6 (March 2014): 938-940.
Abraham, DM, and Wolf, MJ. "Disruption of sarcoendoplasmic reticulum calcium ATPase function in Drosophila leads to cardiac dysfunction. (Published online)" PLoS One 8, no. 10 (2013): e77785-.