Steven D. Crowley, MD, Primary Investigator
The Crowley Lab explores immune-mediated mechanisms of target-organ damage in disease states such as hypertension and kidney fibrosis. Our work has focused on the contribution of type 1 angiotensin receptors in specific tissue pools to kidney and cardiovascular injury.
Recent work from the laboratory has characterized how inflammatory cells modulate end-organ injury and how AT1 receptors on bone marrow-derived cells regulate immune responses in the setting of hypertension.
Nathan Rudemiller, PhD
Undergraduate Student- UNC